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There is no anatomic landmark for the imiquimod between the body and tail of the pancreas although the left border of the aorta is sometimes used to mark the junction (2, 6). Hellman defined the tail as the one fourth of the pancreas from the tip of the tail to the head, whereas Wittingen defined the junction between the body and tail as the point where the gland sharply narrowed (4, 16). It would be difficult to define r e i k i point in the pancreases shown in Figure 1.

Full development of acinar tissue extends into the postnatal period. In mice, pancreatic development begins at embryonic day 8. The ventral diverticulum gives rise to the common bile duct, gallbladder, liver and the ventral pancreatic anlage that becomes a portion of the head of the pancreas with its duct system including the uncinate portion of the pancreas.

The dorsal pancreatic anlage gives rise to a portion of the head, the body, and tail of the pancreas r e i k i a major duct that is continuous through the three regions. The minor papilla that drains the duct of Santorini is derived from the dorsal anlage.

Anatomic variations in the pancreatic and common bile r e i k i systems. The anatomic variations depicted provide additional examples of individual differences in pancreatic anatomy seen in adults. These will be most easily understood by comparing Figures 11 and 12.

It becomes apparent that the duct of Santorini is derived from r e i k i dorsal anlage, whereas the duct of Wirsung (the main duct of the pancreas) is derived from the fusion of duct systems of both dorsal and ventral anlagen and drains into r e i k i duodenum at the ampulla of Vater as depicted in A and B. The connection of the duct of Santorini to the duodenum may regress as depicted in A or persist as in B, C, and D.

Rarely the duct systems may fuse but lose their connection to the ampulla as depicted in D. Pancreatic secretions then reach the duodenum through the duct of Santorini and the minor papilla. Eponymic names identify the anatomist, embryologist or physician who is credited with first describing a structure. You may conclude that Wirsung, R e i k i, and Vater were such scientists. Anatomic variations in the union of the common bile duct and the main pancreatic duct at the major papilla (ampulla of Vater).

The common channel may be long r e i k i Diltiazem Hcl (Tiazac)- FDA in A or short as in B. Less often, there r e i k i no common channel because the ducts open separately into the duodenum as depicted in C. The common channel has received much attention because stones in the biliary tract (gallstones) may lodge in the common channel causing obstruction of both pancreatic and biliary duct systems.

Such an obstruction is frequently regarded as the cause of acute pancreatitis. Figures 14-29 depict the histology of the exocrine pancreas at the light and electron microscopic levels. Most histologic images are from human tissue. Exceptions are usually noted in the legend. For additional ultrastructural detail the reader is referred to the chapter by Kern (8). This tissue section illustrates developing exocrine tissue in the center (arrows) surrounded by primitive mesenchymal Sultrin (Sulfathiazole, Sulfacetamide and Sulfabenzamide)- FDA hematopoietic cells at an estimated gestational age of 5 weeks.

The acinar tissue is composed of a r e i k i of interconnecting tubules. The exocrine pancreas is a Vincristine Sulfate (Vincristine Sulfate Injection)- FDA tubular network.

The point of this drawing is that pancreatic acini are not arranged in clusters like grapes at the ends of a branching duct system but rather as an anastomosing prostate milking massage network that at some termini form classic acini.

Centroacinar cells are typically located at the junction of an acinus or acinar tubule with a small ductule, but they may be interspersed within an acinar tubule.

In this drawing many acinar cells have been replaced by duct cells. This process, called acinar to ductal metaplasia r e i k i, occurs in chronic pancreatitis (3). Acinar cells stain blue at their base enterogermina by sanofi of the high content of RNA and the presence of nuclei. They are pink at their apex (lumenal aspect) where there is a high content of zymogen proteins (digestive enzymes).

The nuclei of centroacinar cells are why do we dream seen within an acinus (arrows). Pancreatic tissue with acinar, centroacinar and ductal cells (EM thick section).

The acinar cells are larger than centroacinar cells and are easily identified because of the darkly stained zymogen granules (ZG). The basal portion (B) of the acinar cells lies next to the interstitial space that contains vessels (V), nerves and connective tissue. Nuclei (N) with nucleoli r e i k i are in the basal portion of the acinar cells.

The golgi (G) lies at the junction of the basal and apical (A) portions of the cell. Centroacinar cells (CAC) have less rough endoplasmic reticulum and no secretory granules. Their cytoplasm is more lightly stained. A small ductule (D) extends from image r e i k i to below center.

The presence of numerous round empty capillaries (arrows) in the interstitial spaces indicates that the pancreas was perfused with fixative. A small branching intralobular duct is evident at the top of the field. Blue zymogen granules are conspicuous in the acinar cells. Acinar and centroacinar cells (low power electron micrograph). Zymogen granules, RER (rough endoplasmic reticulum), and nuclei are all identifiable in the acinar cells.

In addition, several small dense inclusions of variable structure are present in the cytoplasm (lower red arrow). These are residual bodies derived from degradation of acinar cell organelles by lysosomal enzymes. The formation of such residual bodies is called autophagy, and large complex membrane-bound structures reflecting this process are called Cordran Lotion (Flurandrenolide Lotion)- Multum vacuoles.

An acinar lumen is indicated by a small black arrow that lies between two centroacinar cells left of center.

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